Excess Matters

Tag: nutritional therapy

  • The Natural Menopause Method by Karen Newby: Summary, Key Ideas & Review

    Book in one sentence: A BANT-registered nutritional therapist walks you through a food-first, supplement-supported framework for managing menopause symptoms without relying on HRT.

    What Is The Natural Menopause Method About?

    Most menopause books land in one of two places. Either they read like a clinical briefing (all evidence, no warmth) or they drift into vague “eat clean, reduce stress” territory that sounds helpful and means almost nothing. Karen Newby’s book sits in a more useful middle ground.

    Newby is a BANT-registered nutritional therapist with a degree in Nutritional Medicine. Her angle is practical: she wants you to understand the biochemistry well enough to make confident choices, then give you specific food, herb, and supplement interventions tied to each mechanism. The book is not anti-HRT. A menopause specialist contributes a foreword positioning HRT as one valid tool among several, and Newby frames her approach as complementary rather than competing. That framing matters, because it keeps the book usable for women across a wide range of circumstances.

    What sets this apart from generic wellness content is the specificity. Newby explains why declining oestrogen produces hot flushes (it disrupts insulin sensitivity and triggers adrenaline surges), why sleep unravels (oestrogen supports serotonin, which is the precursor to melatonin; progesterone supports GABA, the brain’s calming neurotransmitter), and why constipation is a hormonal issue rather than just a digestive inconvenience. Once you know the mechanism, the food recommendations stop feeling arbitrary.

    The Four Shifts: How Newby Structures the Approach

    The book’s backbone is a sequenced protocol called the Four Shifts. Each shift addresses a different physiological layer, and the order matters.

    Shift 1: Reset comes first because of something most women don’t know: the adrenal glands are the body’s backup source of both oestrogen (as the weaker form, oestrone) and progesterone when the ovaries start to wind down. Chronic stress means those same adrenal glands are busy prioritizing cortisol instead. As Newby puts it: “Stress (survival) trumps sex hormones.” Addressing cortisol load before anything else is not a soft wellness suggestion. It is a physiological prerequisite.

    Shift 2: Cleanse focuses on the liver and gut as an integrated oestrogen clearance system. The liver converts oestrogen into less active forms; the gut eliminates them. Disruptions anywhere in this pathway (poor diet, constipation, low microbiome diversity) cause processed oestrogen to be reabsorbed rather than excreted, raising total oestrogen load even as the ovaries produce less. Newby calls this the estrobolome effect, and her interventions address both ends simultaneously: brassicas daily for liver support, fermented foods and ground linseed for gut elimination.

    Shift 3: Rest maps specific food and supplement strategies to the three clinical sleep failure modes she sees in her practice (trouble falling asleep, trouble staying asleep, waking exhausted). Tryptophan-rich foods support serotonin and melatonin production. Magnesium supports GABA. Avoiding tyramine-rich foods near bedtime (cheese, cured meats, wine, chocolate) prevents noradrenaline spikes that keep the brain alert.

    Shift 4: Eat optimizes phytoestrogen intake and nutrient density. This is Newby’s “sparkplug” model: macronutrients are the fuel, micronutrients are the sparkplugs. A car will not run without both. The final shift covers the therapeutic phytoestrogen protocol, whole-food swaps, and supplement quality guidelines.

    The shifts are sequential but not rigid. A woman with severe insomnia might start with Shift 3. The framework is a map, not a prescription, and Newby’s repeated framing is “consistency over perfection.”

    Why Does Blood Sugar Keep Coming Up in a Menopause Book?

    It comes up because it is everywhere. Blood sugar instability is the single highest-leverage variable in the perimenopause symptom picture, and Newby returns to it in nearly every section.

    Here is the short version of the mechanism. Oestrogen helps regulate insulin sensitivity. As oestrogen declines, cells become less responsive to insulin. Foods that produced stable energy at thirty-five now create larger glucose swings at forty-five. Those swings trigger cortisol and adrenaline (already overtaxed at perimenopause). In vasomotor terms, a glucose low triggers an adrenaline surge that causes vasodilation, which is how blood sugar directly drives hot flush frequency. In mood terms, the same low amplifies anxiety, irritability, and the impulse to eat something immediately.

    Newby’s practical protocol is not complicated:

    • A 12-14 hour overnight fast (nothing exotic, just not eating at 10pm)
    • Protein and fat at every meal to slow glucose absorption
    • Never skip breakfast (which extends the cortisol spike from overnight fasting)
    • Caffeine only with food (on an empty stomach, caffeine puts the body into fight-or-flight and raises cortisol directly)

    “I liken sugar to pouring petrol onto a fire — the flames burn really bright and kick out a lot of heat, which can give us a sense of energy; but after this short spike the flames become even smaller than they were before. Putting protein and good fats on the fire I liken to coal — although the flames don’t burn as brightly, more heat is produced and they burn for longer.”

    The food swap table in this section is among the more practically useful pages in the book. The 3pm coffee-and-biscuit ritual (which Newby notes works partly through habituated dopamine cues, not hunger) gets replaced with fresh mint tea, miso soup, tamari seeds, or falafel with hummus. These are crowding-out strategies rather than restrictions.

    She also brings the emotional eating angle into this framework. Physical hunger builds gradually, involves stomach grumbling (the hormone ghrelin), and is resolved by eating. Emotional hunger arrives suddenly, is unrelated to the last meal, and is not resolved by eating (which is why the craving continues after the food is gone). The Japanese have a word for it: kuchisabishii, meaning “lonely mouth.” The dopamine reward system drives craving behavior regardless of hunger state, and ultra-processed foods are engineered to spike that system. Knowing this does not eliminate the craving, but it reframes what is happening: it is a neurochemical response to a product designed to produce it, not a character flaw.

    What About the Weight Changes?

    Weight gain during perimenopause, especially around the middle, follows a specific hormonal logic that Newby explains clearly. As oestrogen declines, the pattern of fat storage shifts from hip and thigh to abdominal, which is a testosterone-dominant pattern. The abdominal fat itself then converts testosterone to oestrogen (through an enzyme called aromatase), which can raise oestrogen load even as the ovaries produce less, creating a feedback loop.

    Phytoestrogens are Newby’s sharpest tool for addressing this pattern directly. These are plant compounds structurally similar to oestradiol that bind to oestrogen receptor sites and modulate them bidirectionally: reducing symptoms from oestrogen excess and relieving symptoms from oestrogen deficiency. NICE guidelines confirm that isoflavones may relieve vasomotor symptoms. Research also supports their role in bone density, memory function, and reduced oxidative stress.

    The three main sources:

    • Isoflavones (soya in cooked or fermented forms): tofu, tempeh, miso, natto, edamame; also chickpeas, lentils, peas
    • Lignans: ground linseed or flaxseed (the highest dietary source), sesame seeds, cashews, brassicas, apples, apricots, cherries
    • Coumestans: soybean sprouts, alfalfa, split peas, pinto beans

    Two practical rules stand out. Cook or ferment soya before eating it (raw lectins may affect iodine uptake and are deactivated by heat and fermentation). Fermented foods also supply the lactic acid needed to absorb phytoestrogens in the first place, which is why kefir, sauerkraut, and miso appear throughout the protocol.

    On the supplement side, Newby’s guidance is quality-first: the form of the mineral matters as much as the dose. Magnesium glycinate or malate over oxide. Zinc citrate or picolinate over oxide. Calcium citrate over carbonate. Many supermarket supplements contain fillers, glycerol, sucrose, and talc, so reading the ingredients list matters more than reading the nutrient label.

    Sage (as herb, tea, or tablet) gets specific mention as an evidence-backed hot flush intervention: research supports reductions in both frequency and severity. Red clover isoflavone supplements similarly have research backing for vasomotor symptoms and mood.

    Is The Natural Menopause Method Worth Reading?

    Read this if you are in perimenopause or approaching it, want to understand the mechanisms behind your symptoms, and are willing to make incremental food-based changes over time. Women who have found generic “eat clean” advice unhelpful will get more traction here because Newby explains the biochemistry behind each recommendation. Women who are not on HRT (by choice, contraindication, or circumstance) will find a comprehensive food-first toolkit that few books in this category match. Women who are on HRT will still find value in the lifestyle layer.

    Skip it if you want a meal plan with precise macros, you are in North America and find UK supplement brands frustrating to source (the food interventions translate; the brand names do not), or you prefer narrative-driven health books (parts of this read more like a clinical reference).

    One caveat: the book covers an enormous amount of territory (biochemistry, recipes, pelvic floor rehabilitation, acupuncture, supplement protocols) in 256 pages. Some sections feel compressed as a result. The supplement lists in particular can feel overwhelming without a background in nutritional therapy. Start with the food interventions and treat the supplement section as a reference to return to.

    Books Like The Natural Menopause Method

    BookAuthorBest For
    The Natural Menopause PlanMaryon StewartBroader lifestyle approach with HRT alternatives
    Eat to Thrive During MenopauseJenn Salib HuberAnti-diet framework with intuitive eating integration
    Healthy HormonesMagdalena WszelakiHormone-balancing nutrition with lab-tested protocols
    The Menopause CompanionKathleen DaviesIntegrative approach covering conventional and natural options
    The Happy Hormone GuideShannon LeparskiPlant-based hormone support with cycle-syncing emphasis

  • The Craving Cure by Julia Ross: Summary, Key Ideas & Review

    The book in one sentence: A clinical psychologist and nutritional therapy pioneer argues that food cravings are caused by neurotransmitter deficiencies, and that targeted amino acid supplements can stop them, often within 24 hours.

    What Is The Craving Cure About?

    You’ve probably tried the willpower version. You white-knuckle through the afternoon, eat the sad salad, feel proud of yourself for exactly four hours, and then eat a sleeve of crackers at 9 PM. The next morning, you circle back to the same explanation you always land on: something is wrong with you.

    Julia Ross has a different theory. She spent more than thirty years running addiction and eating disorder clinics in the San Francisco Bay Area (over four thousand clients), and what she observed was consistent: cravings are not a failure of character. They are a symptom of measurable brain chemistry deficits. When five key neurotransmitter systems run low, the brain generates involuntary drives toward processed sugar and starch. Not because you’re weak. Because your brain is trying to self-medicate with the only fast-acting chemicals it has access to.

    Ross is also a psychotherapist, which matters for how she argues. She’s not dismissing the emotional dimension of eating. She’s saying that until the brain’s depleted chemistry is restored, no amount of insight, therapy, or resolve will reliably stop the cravings. The sequence she proposes is: fix the brain first, then everything else becomes possible. The Craving Cure is her 432-page clinical manual for how to do that.

    Why Do You Crave? The Neurotransmitter Deficiency Model

    Picture the brain’s appetite regulation as a five-instrument orchestra. When all five are playing well, you eat when you’re hungry, stop when you’re full, and don’t think much about food otherwise. When any one instrument falls out of tune, the result is cravings.

    Ross calls those five systems “the Fabulous Five”: serotonin, blood glucose stability, endorphins, GABA, and the catecholamines (dopamine and norepinephrine). Each system, when depleted, generates a specific and predictable craving pattern. The mechanism isn’t mysterious. Serotonin drops in the afternoon as daylight fades, so Type 1 cravers reliably want carbs at 4 PM. GABA depletes under chronic stress, so Type 4 cravers reach for salty, crunchy foods when they’re overwhelmed. The cravings aren’t random. They’re the brain’s precise, involuntary attempt to restore what’s missing.

    The deeper problem is how processed foods made this worse. Starting in the 1970s, Ross identifies three dietary shifts that simultaneously depleted neurotransmitter-building nutrients and flooded the food supply with substances that exploit the brain’s reward systems: the replacement of animal fats with processed vegetable oils, the explosion of refined sugar and high-fructose corn syrup, and the cultural slide away from animal protein. Before 1970, fewer than a third of Americans were overweight. That’s not nostalgia for Ross. It’s evidence that the current epidemic is caused, not inevitable.

    “Knowing more, willing more, eating less — these strategies are simply no match for the avalanche of pleasure that our Techno-Karbz can trigger in the brain. What else could regularly overwhelm the good intentions of 230 million adults?”

    If 230 million people keep failing at the same task using the same strategies, the strategies are wrong. That’s the whole argument in one move.

    The 5 Craving Types: Which One Are You?

    The Craving-Type Questionnaire (developed over thirty years and twenty thousand amino acid trials) takes about ten minutes and maps your symptom patterns to specific neurotransmitter deficiencies. Most people have more than one type. Here’s what each looks like in practice:

    Type 1: The Depressed Craver (Low Serotonin)

    Cravings hit hardest in the afternoon, evening, and winter. Mood symptoms tag along: negativity, anxiety, worry, perfectionism, insomnia, a low-grade irritability that gets worse as the day goes on. The late-night cereal, the 4 PM chocolate, the “I just need something sweet before I can sleep” (all serotonin). The brain is reaching for carbohydrates because carbs temporarily boost tryptophan’s access to the brain. The amino acid fix is tryptophan or 5-HTP, taken as needed.

    Type 2: The Crashed Craver (Blood Sugar Instability)

    Skip breakfast, feel fine until 10 AM, then raid the office candy bowl like your life depends on it. Or go too long between meals, get shaky and foggy, and make a fast-food decision you’ll regret. This type isn’t technically a neurotransmitter deficiency. It’s a fuel crisis. The brain has no stored glucose and demands a continuous supply. L-glutamine dissolved under the tongue can substitute for glucose in the brain and stop the crash-and-crave episode within minutes.

    Type 3: The Comfort Craver (Low Endorphins)

    This one is about chocolate, creamy textures, doughy foods, and sometimes alcohol. Endorphins are the brain’s endogenous opiates (thousands of times more powerful than morphine at their peak). A University study found M&Ms raised enkephalin activity by 150%, comparable to opium. Ross is not using “addiction” loosely here. The person who says “it’s my one pleasure” and genuinely means it, who feels a small grief at the thought of giving up chocolate, is describing an endorphin deficit. DPA and DLPA work by slowing the enzymes that break down natural endorphins, raising their levels without adding external opioids, without tolerance, without dependence.

    Type 4: The Stressed Craver (Low GABA)

    Chips at the desk. Crackers by the handful. GABA is the brain’s primary inhibitory neurotransmitter, the biochemical antidote to adrenaline. It’s what lets you decompress after a hard day. When it’s depleted by chronic stress and a protein-poor diet, salty, crunchy, starchy foods provide a brief, unsatisfying simulation of the calm the brain is missing. GABA is the fastest-acting of all the interventions: chewed in tablet form (Ross recommends 125 mg), it can produce visible neck and shoulder relaxation within seconds.

    Type 5: The Fatigued Craver (Low Catecholamines)

    Triple espresso people. Energy drink people. “I cannot function without coffee” people. Dopamine and norepinephrine are the brain’s natural stimulants, and when they’re low, food becomes a stimulant delivery system (not comfort or pleasure, but energy). Tyrosine, the direct precursor to the catecholamine family, typically restores energy and focus within five to ten minutes. Ross includes a detailed caffeine withdrawal protocol built around tyrosine replacing the energy effect cup by cup.

    How Amino Acid Therapy Works (and How Fast)

    The protocol has two phases, and the order matters.

    Phase one is the amino acids. You identify your craving type, start the indicated supplements, and within one to seven days (often within hours) the cravings diminish. Ross’s clients consistently report the same thing after their first day: “Amazing. My cravings disappeared.” The mechanism isn’t magic. Amino acids are precursors to neurotransmitters, and the brain can upregulate production relatively quickly once the raw material is available. The speed of response also functions as a diagnostic tool: if the amino doesn’t help, you’ve identified the wrong type.

    Phase two is the food plan. Ross calls it the Primal Plate, a return to pre-1970s eating built around adequate animal protein (the primary dietary source of all five key amino precursors), traditional fats, and the elimination of processed sugar and flour. The food plan is not calorie-restricted. Low-calorie dieting, she argues, starves the brain of protein and deepens neurotransmitter depletion, making cravings worse. Typically, after two to twelve months on the protocol, clients can stop the supplements entirely. The food becomes the chemistry.

    The reason the phases can’t be reversed matters. Attempting to change your diet while your brain chemistry remains depleted guarantees failure. The depleted brain generates cravings stronger than dietary resolve. The amino acids buy time and demonstrate what craving-free life feels like (experientially, not just conceptually). When someone who has blamed themselves for decades feels their food compulsion dissolve within twenty minutes of the right amino acid, the reframe from “willpower failure” to “brain chemistry” becomes something they’ve lived, not just read.

    One honest note on evidence: the amino acid protocols are primarily supported by Ross’s thirty years of clinical observation and the broader neuroscience literature on neurotransmitters, not randomized controlled trials. The clinical mechanism is solid; the RCT base is thin. This is worth knowing before you build a supplement stack around her recommendations.

    Is The Craving Cure Worth Reading?

    Read this if you have cravings that feel genuinely compulsive (not “I’d enjoy a cookie” but “I cannot get through the afternoon without this”), if your cravings follow distinct patterns tied to time of day, season, stress level, or skipped meals, or if you’ve done real psychological work on your relationship with food and found it clarifying but insufficient. The neurotransmitter framework applies to mood and eating simultaneously, which makes it useful for anyone with depression, anxiety, or chronic fatigue sitting alongside their food difficulties.

    Skip it if you have a history of restrictive eating disorders. The elimination of processed foods and the firm categorization of what’s permissible can amplify restriction patterns in ways the book doesn’t adequately address. Ross’s intended audience is compulsive overeaters, not restrictors, and the book doesn’t make that distinction clearly enough. Also skip it if you’re currently working within an intuitive eating framework — food rules and rebuilding interoceptive trust don’t mix well.

    One caveat: the book is 432 pages, and roughly half of that is the dietary framework and recipes rather than the amino acid protocol. The core clinical protocol is in the first 150 pages. The rest is useful if you’re going all-in on the Primal Plate, but don’t let the length put you off the material that matters.

    Books Like The Craving Cure

    BookAuthorBest For
    The End of OvereatingDavid KesslerUnderstanding how the food industry engineered your vulnerability — the external mechanism Ross’s book treats
    The Mood CureJulia RossSame amino acid framework applied to depression, anxiety, and trauma rather than food cravings
    The Hunger HabitJudson BrewerMindfulness-based approach to cravings — different mechanism (reward-based learning), complementary goal
    Bright Line EatingSusan Peirce ThompsonArrives at similar dietary conclusions (eliminate sugar and flour) through behavioral architecture instead of amino acid repair
    The End of CravingMark SchatzkerNutritive mismatch theory — processed food trains the brain to decouple taste from nutrition, creating endless craving